In a nutshell: Drug-eluting stents increase risk of coronary artery thrombosis (clot). Drug-eluting stents reduce collateral vessel growth (vascularization). Vessel growth helps the heart handle the loss of blood flow if a clot occurs in the stent site in the coronary artery. So patients with drug-eluting stents not only are more likely to develop a clot, but for that clot to cause more damage due to reduced vascularization in the area of the clot.
Doctors suggest Plavix and aspirin treatment for at least a year to prevent thrombosis (clots).
Drug-eluting Stents Reduce Collateral Vessel Growth - New study indicates that drug-eluting stents may obstruct the heart's natural ability to form small collateral blood vessels - . For patients with coronary artery blockages, these vessels normally help supply blood to areas starved by artery blockages. The drug-eluting stents appear to hinder this natural, healing process.
Drug-eluting stents . So when patients stop Plavix, clots form (due to exposed metal in artery lining from stent?).
One man, as prescribed by his doctor, s. I wonder: When the drug is gone from the drug-eluting stent (after 6 months?), will the artery lining form properly? Did not appear to do so with that 41 year-old man. If not, is Plavix is safe to take for long-term? Scientists speculate that the problems arise when these drug-eluting stents are used incorrectly, in vessels that are too narrow or where blockages too extensive.
Angioplasty & Stents Post Heart Attack - This was a little unexpected. Study indicates that using angioplasty or inserting stents more than 25 hours after a heart attack does NOT increase survival rates.
Where's the logic in that?
This article doesn't attempt to explain--in fact, doctors are perplexed by the result--but the piece is still worth reading.